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In multiple comparisons of age variables, the differences between the MDD group and the control group and between the MDD group and the remission group were significant, but there was no difference between the control group and the remission group (Table 1)
In white adipose tissue of obese, insulin-resistant and aged mammals, NNMT is dramatically upregulated and because it consumes both the universal methyl donor (SAM) and the NAD precursor (nicotinamide), its hyperactivity simultaneously depletes the cellular methylation reserve and the NAD-salvage pool [1,2]
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